[TheCruxofBag]

Churu, Rajasthan/Suryapet, Telangana

In a gym in Churu, a 49-year-old high school principal, Birbal M., was running on a treadmill when he suddenly collapsed. He was given CPR by the gym operator before being taken to hospital, where he was declared dead. Doctors believe he suffered a heart attack while on the treadmill.

Meanwhile, at a marketplace in Suryapet, Bezawada S. was buying groceries when he suffered a fatal heart attack.

[Mr.NoCredit]

Damn! They're dropping like flies over there.

If they leave those bodies lying around there’ll be a lot more flies..

[Necrotic Bowel]

In both videos they prop the poor SOB upright, thereby assuring a decrease in blood flow to the brain. Bad idea. Lay them on their back and start CPR. That’s what you’re supposed to do. But it’s India so that’s not gonna happen. They take more of a “walk it off” approach to the treatment of acute myocardial infarction.

How the hell are there still a billion of them, anyway? All they must do is fuck.

[DiabeticEric]

It has to be all the sodium in their food.

[Mmichael]

the smell of a thousand armpits inside that hellish gym

[DarkSighted]

2nd vid: just a ploy to steal some apples and cop a feel, poorly 'executed'. :D

[kellyhound]

According to doctors at top institutions like Apollo Hospitals and AIIMS, heart disease strikes South Asians 10 to 15 years earlier than people in Western countries.

South Asians have a high genetic predisposition to premature atherosclerosis. South Asians are genetically prone to carrying higher levels of Lipoprotein(a), which is an inherited type of cholesterol particle. Unlike standard LDL, the bad cholesterol, Lp(a) is highly sticky and prone to causing blood clots.

It accelerates the formation of hard plaques inside the coronary arteries at a very early age. Standard lipid blood tests often miss elevated Lp(a), leaving young individuals unaware of their risk.

Standard lipid blood tests miss elevated Lipoprotein(a) [Lp(a)] because they only measure generic categories of cholesterol, such as total, LDL, HDL, and Triglycerides, rather than the specific, genetically determined protein structures that form Lp(a).

Because Lp(a) requires a distinct, specialized blood assay, millions of people, including young individuals with perfect routine cholesterol scores, remain entirely unaware of their elevated risk.

Because Lp(a) levels are 80-90% determined by genetics, standard healthy lifestyle habits do not lower or alter them.

Most global medical guidelines historically recommended screening only for individuals with an established family history of premature heart disease. As a result, less than 5% of the population has ever had their levels checked. To see it, a clinician must specifically check a completely separate biomarker box on the lab slip: the Lipoprotein(a) test (CPT code 83695).

Major organizations like the American Heart Association now advocate that every adult should be tested at least once in their lifetime to eliminate this blindspot.

Standard cholesterol drugs like statins do not effectively lower Lp(a). Doctors cannot prescribe a pill specifically designed to eliminate Lp(a), instead, if a patient tests high, the current strategy is aggressive risk management, lowering standard LDL even further, managing blood pressure and optimizing lifestyle to compensate for the genetic risk.

An elevated Lp(a) level is medically defined as 50 mg/dL (or 125 nmol/L) and above. The distribution across populations highlights why South Asians are at a disproportionate risk.


~20% (1 in 5 Americans) which translates to roughly 63 million people in the U.S. Despite this, less than 1% of Americans have ever had it checked. Prevalence peaks significantly higher among Black Americans.


25% to 35% ( 1 in 3 Indians). South Asians have some of the highest baseline levels globally. Shockingly, studies show that up to 34% of Indian patients admitted with acute heart failure or coronary syndromes carry dangerously high Lp(a).

Also wrote before that cardiologists note a genetic compounding effect. The exact same elevated level of Lp(a) that increases heart disease risk by 1.5 times in Western populations increases the risk by 3 times in Indians due to their smaller, more fragile coronary arteries.

While a standard test measures the weight of LDL cholesterol, ApoB measures the exact number of plaque-causing particles in your blood.

Every single plaque-forming cholesterol particle (including LDL, VLDL, and Lp(a)) carries exactly one molecule of ApoB.

South Asians frequently have a high number of small, dense LDL particles. A standard test might report a normal LDL weight, but an ApoB test will reveal that you actually have a dangerously high number of these tiny, artery-penetrating particles.

Cholesterol alone does not cause heart attacks as it requires inflammation to turn into unstable, rupture-prone plaque.

Due to a genetic tendency toward visceral fat storage and metabolic stress, South Asians naturally exhibit higher baseline vascular inflammation. An hs-CRP level above 2.0 mg/L indicates elevated cardiac risk.

Rather than just checking standard fasting glucose, measuring fasting insulin evaluates how hard your pancreas is working.

Insulin resistance damages the delicate inner lining (endothelium) of blood vessels years before diabetes officially develops.

Due to the Asian Indian Phenotype, South Asians can develop severe insulin resistance at a completely normal weight or low BMI.

In addition to blood markers, cardiologists strongly recommend a Coronary Artery Calcium (CAC) Scan for South Asians over the age of 30–35 if they have a family history of early heart disease.

This is a quick, low-dose chest CT scan that takes a physical picture of your heart arteries to check if hard calcium plaque has already started building up, allowing for early, life-saving medical intervention.

The traditional South Asian diet is often heavy in refined carbohydrates (white rice, roti, maida) and deep-fried snacks, which spikes insulin and increases small, dense LDL particles (ApoB).

Chronic (work) stress releases high amounts of cortisol and adrenaline, which constricts blood vessels, raises blood pressure, and causes the liver to release extra glucose, directly worsening insulin resistance especially for workers in high-stress, sedentary environments (like IT, finance or corporate roles).

Smoking is the most destructive, preventable trigger for sudden heart attacks, especially in South Asians who already carry a genetic risk. While genetics lay the groundwork for plaque buildup, smoking acts as the match that ignites the fire.

Many young people turn to alternatives thinking they are safer for the heart, but clinically, this is a misconception.

Vaping/E-cigarettes: While they lack tobacco tar, they still deliver high doses of nicotine and chemical particulates. Studies show vaping causes immediate arterial stiffness and endothelial dysfunction similar to traditional cigarettes.

Hookah / Shisha: A single one-hour hookah session can expose a user to as much smoke and carbon monoxide as inhaling 100 or more cigarettes, causing massive oxidative stress to the heart muscles.

Smokeless Tobacco (Gutka, Khaini, Paan): Highly prevalent in South Asia, these deliver massive, sustained rushes of nicotine directly into the bloodstream, spiking blood pressure and damaging blood vessels continuously.

Also the genetic tendency to store visceral fat (fat around the organs) is incredibly dangerous for a 45-year-old corporate worker, but historically, it was the exact reason children survived to adulthood.

During the devastating famines that hit the Indian subcontinent over the last 10,000 years, childhood mortality was exceptionally high.

Children carrying the thrifty gene could store energy out of almost nothing, allowing them to survive winters, droughts and crop failures.

The individuals who lacked these genes died in childhood and never reproduced. Therefore, the modern South Asian population is exclusively made up of the descendants of those master-survivors.

In evolutionary biology, this is known as an evolutionary trade-off. The very genes that drastically lowered the risk of dying during childhood now cause a much higher risk of dying after childhood (specifically during middle age, from their 30's to 50's).

Here comes the corona shots debunking (again).

Following public anxiety over these earlier deaths, major research bodies, including the Indian Council of Medical Research and the All India Institute of Medical Sciences conducted extensive, year-long autopsy and patient studies.

Their data showed no statistically significant association between COVID-19 vaccination status and sudden unexplained deaths in individuals aged 18 to 45.

Post-mortem analyses proved that these sudden deaths were overwhelmingly caused by advanced, underlying coronary artery disease (atherosclerosis), and not vaccine complications.

While the vaccine is safe, the actual COVID-19 virus itself did leave a lasting cardiac impact.

Contracting a severe case of COVID-19 is proven to cause severe vascular inflammation, endothelial damage, and an increased tendency for blood clotting. For some individuals, this post-viral damage can make pre-existing arterial plaques more unstable.

Global cardiology data shows that getting vaccinated actually provides a cardioprotective benefit, significantly lowering your long-term risk of a virus-induced stroke or heart attack.

Sudden cardiac death among young people with underlying genetic defects or severe blockages has always occurred, but it used to happen privately and nowadays there are cameras everywhere.

Bezawada S was 36 years old.

[DavidAaronGarcia]

Originally Posted by kellyhound ↗

According to doctors at top institutions like Apollo Hospitals and AIIMS, heart disease strikes South Asians 10 to 15 years earlier than people in Western countries.

South Asians have a high genetic predisposition to premature atherosclerosis. South Asians are genetically prone to carrying higher levels of Lipoprotein(a), which is an inherited type of cholesterol particle. Unlike standard LDL, the bad cholesterol, Lp(a) is highly sticky and prone to causing blood clots.

It accelerates the formation of hard plaques inside the coronary arteries at a very early age. Standard lipid blood tests often miss elevated Lp(a), leaving young individuals unaware of their risk.

Standard lipid blood tests miss elevated Lipoprotein(a) [Lp(a)] because they only measure generic categories of cholesterol, such as total, LDL, HDL, and Triglycerides, rather than the specific, genetically determined protein structures that form Lp(a).

Because Lp(a) requires a distinct, specialized blood assay, millions of people, including young individuals with perfect routine cholesterol scores, remain entirely unaware of their elevated risk.

Because Lp(a) levels are 80-90% determined by genetics, standard healthy lifestyle habits do not lower or alter them.

Most global medical guidelines historically recommended screening only for individuals with an established family history of premature heart disease. As a result, less than 5% of the population has ever had their levels checked. To see it, a clinician must specifically check a completely separate biomarker box on the lab slip: the Lipoprotein(a) test (CPT code 83695).

Major organizations like the American Heart Association now advocate that every adult should be tested at least once in their lifetime to eliminate this blindspot.

Standard cholesterol drugs like statins do not effectively lower Lp(a). Doctors cannot prescribe a pill specifically designed to eliminate Lp(a), instead, if a patient tests high, the current strategy is aggressive risk management, lowering standard LDL even further, managing blood pressure and optimizing lifestyle to compensate for the genetic risk.

An elevated Lp(a) level is medically defined as 50 mg/dL (or 125 nmol/L) and above. The distribution across populations highlights why South Asians are at a disproportionate risk.


~20% (1 in 5 Americans) which translates to roughly 63 million people in the U.S. Despite this, less than 1% of Americans have ever had it checked. Prevalence peaks significantly higher among Black Americans.


25% to 35% ( 1 in 3 Indians). South Asians have some of the highest baseline levels globally. Shockingly, studies show that up to 34% of Indian patients admitted with acute heart failure or coronary syndromes carry dangerously high Lp(a).

Also wrote before that cardiologists note a genetic compounding effect. The exact same elevated level of Lp(a) that increases heart disease risk by 1.5 times in Western populations increases the risk by 3 times in Indians due to their smaller, more fragile coronary arteries.

While a standard test measures the weight of LDL cholesterol, ApoB measures the exact number of plaque-causing particles in your blood.

Every single plaque-forming cholesterol particle (including LDL, VLDL, and Lp(a)) carries exactly one molecule of ApoB.

South Asians frequently have a high number of small, dense LDL particles. A standard test might report a normal LDL weight, but an ApoB test will reveal that you actually have a dangerously high number of these tiny, artery-penetrating particles.

Cholesterol alone does not cause heart attacks as it requires inflammation to turn into unstable, rupture-prone plaque.

Due to a genetic tendency toward visceral fat storage and metabolic stress, South Asians naturally exhibit higher baseline vascular inflammation. An hs-CRP level above 2.0 mg/L indicates elevated cardiac risk.

Rather than just checking standard fasting glucose, measuring fasting insulin evaluates how hard your pancreas is working.

Insulin resistance damages the delicate inner lining (endothelium) of blood vessels years before diabetes officially develops.

Due to the Asian Indian Phenotype, South Asians can develop severe insulin resistance at a completely normal weight or low BMI.

In addition to blood markers, cardiologists strongly recommend a Coronary Artery Calcium (CAC) Scan for South Asians over the age of 30–35 if they have a family history of early heart disease.

This is a quick, low-dose chest CT scan that takes a physical picture of your heart arteries to check if hard calcium plaque has already started building up, allowing for early, life-saving medical intervention.

The traditional South Asian diet is often heavy in refined carbohydrates (white rice, roti, maida) and deep-fried snacks, which spikes insulin and increases small, dense LDL particles (ApoB).

Chronic (work) stress releases high amounts of cortisol and adrenaline, which constricts blood vessels, raises blood pressure, and causes the liver to release extra glucose, directly worsening insulin resistance especially for workers in high-stress, sedentary environments (like IT, finance or corporate roles).

Smoking is the most destructive, preventable trigger for sudden heart attacks, especially in South Asians who already carry a genetic risk. While genetics lay the groundwork for plaque buildup, smoking acts as the match that ignites the fire.

Many young people turn to alternatives thinking they are safer for the heart, but clinically, this is a misconception.

Vaping/E-cigarettes: While they lack tobacco tar, they still deliver high doses of nicotine and chemical particulates. Studies show vaping causes immediate arterial stiffness and endothelial dysfunction similar to traditional cigarettes.

Hookah / Shisha: A single one-hour hookah session can expose a user to as much smoke and carbon monoxide as inhaling 100 or more cigarettes, causing massive oxidative stress to the heart muscles.

Smokeless Tobacco (Gutka, Khaini, Paan): Highly prevalent in South Asia, these deliver massive, sustained rushes of nicotine directly into the bloodstream, spiking blood pressure and damaging blood vessels continuously.

Also the genetic tendency to store visceral fat (fat around the organs) is incredibly dangerous for a 45-year-old corporate worker, but historically, it was the exact reason children survived to adulthood.

During the devastating famines that hit the Indian subcontinent over the last 10,000 years, childhood mortality was exceptionally high.

Children carrying the thrifty gene could store energy out of almost nothing, allowing them to survive winters, droughts and crop failures.

The individuals who lacked these genes died in childhood and never reproduced. Therefore, the modern South Asian population is exclusively made up of the descendants of those master-survivors.

In evolutionary biology, this is known as an evolutionary trade-off. The very genes that drastically lowered the risk of dying during childhood now cause a much higher risk of dying after childhood (specifically during middle age, from their 30's to 50's).

Here comes the corona shots debunking (again).

Following public anxiety over these earlier deaths, major research bodies, including the Indian Council of Medical Research and the All India Institute of Medical Sciences conducted extensive, year-long autopsy and patient studies.

Their data showed no statistically significant association between COVID-19 vaccination status and sudden unexplained deaths in individuals aged 18 to 45.

Post-mortem analyses proved that these sudden deaths were overwhelmingly caused by advanced, underlying coronary artery disease (atherosclerosis), and not vaccine complications.

While the vaccine is safe, the actual COVID-19 virus itself did leave a lasting cardiac impact.

Contracting a severe case of COVID-19 is proven to cause severe vascular inflammation, endothelial damage, and an increased tendency for blood clotting. For some individuals, this post-viral damage can make pre-existing arterial plaques more unstable.

Global cardiology data shows that getting vaccinated actually provides a cardioprotective benefit, significantly lowering your long-term risk of a virus-induced stroke or heart attack.

Sudden cardiac death among young people with underlying genetic defects or severe blockages has always occurred, but it used to happen privately and nowadays there are cameras everywhere.

Bezawada S was 36 years old.

Basically , in better terms , we're just all ticking time bombs, no, I'm not talking about ISIS, I'm talking about our hearts. We don't know when we're gonna drop dead. By no reality is when I suffer one of my strokes, I practically did drop dead, but I didn't lose conscious, everything froze, and I went and dropped dump.\n There was no camera, of course where I was at Yeah, I obviously recovered through The Strokes. I'm on here leaving these comments got past. The stroke. Stay didn't find any blood clots, but I did suffer heart failure at about 20 to 30% function, which is pretty much nearly nothing but still working. It's scary You just never know when it's gonna hit, also, if you ever end up in something like this with somebody make sure you get them in the hospital within the first 15 minutes, it's very important and hope that they have a good emergency room system there, even in a modern country, like United States of America, I got discharged right away, cause they figured there's nothing wrong. Even though I was wobbling out the hospital Nat's hearing Fresno, California, most people probably already know what hospital that is if you're from this area.\n That being said, that's the emergency rooms or like that, almost at all the hospitals here in this city. I don't know about India where these incidents happen. Are any other parts of this country in America or India? But it's very scary now. I should say the hospital went to they did have good specialist doctors but the catches you have to make it that far. The second stroke, when I faded out, ended up back in the halls film. They started looking at everything a bit more serious. Then that's when I made it to the good specialist. But the catch is I had to make it to that. And that being that's like playing Russian role, that's dangerous. Nobody does something that crazy. I'm almost certainly gonna find a few videos on here due to this website. I guarantee that they're there. Do not do so otherwise do not do that. I'm not saying don't watch the videos but not do not play that game. But it's important, of course, the first 15 minutes to get medical attention due to a heart attack or a stroke.\n The guy's in the second video were on top of it, at least they were performing CP. R on him, and it looks like they were carting him off to a vehicle to get medical attention. Which was smart than knowing how fast it took the ones on the first video